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doi: 10.3390/microorganisms11040967 110 GuptaNAbd El-GawaadNSOsman AbdallahSAAl-DossariM

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Its working mechanism is allosteric facilitation of HGF/c-Met signaling: Dihexa binds HGF with high affinity (reported K d ~65 pM in the original work) and is proposed to potentiate HGF dimerization and c-Met phosphorylation at subthreshold endogenous HGF concentrations

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Therefore, NNMT is expected to act as an effective cancer therapeutic target in BRCA1 -deficient ovary tumor ( Figure 1 NNMT is also involved in regulating autophagy to maintain cancer cell survival via the mTOR and AMPK pathways

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