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Clarifying these processes is essential for understanding the regulatory network of ferroptosis and identifying potential therapeutic targets (Fig

The recovery of endogenous vasopressin levels, which decrease 6 h after septic shock onset [44] but increase at 24 h critical illness [45], may have contributed to this

678,684,688 Alcohol metabolism leads to increased lipid accumulation, depletion of the antioxidant glutathione, and mitochondrial dysfunction through ROS-induced oxidative stress

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