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These results indicate that NRF2 might be compensatory binding to more sites in the absence of SNF2L, possibly attempting to maintain SLC7A11 transcription and redox homeostasis

78 Nevertheless, the potential risks and benefits of cytokine inhibition must be carefully addressed to guide the continuation or halting of such treatments

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IL-22(+)CD4(+) T cells promote colorectal cancer stemness via STAT3 transcription factor activation and induction of the methyltransferase DOT1L

Sigma) containing Ca 2+ and Mg 2+