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One of the key reasons that CSCs remain difficult to eliminate is their intrinsic plasticity, allowing them to transition between quiescent and proliferative states, adopt epithelial or mesenchymal phenotypes, and reprogram their metabolic and epigenetic landscapes in response to therapeutic stress

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Molecular weight: approximately 1419.5 Da

10.1016/j.aquatox.2017.07.005 40

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Mapping and refactoring pathway control through metabolic and protein engineering: the hexosamine biosynthesis pathway