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To understand why senescent cells are more prone than normal counterparts to undergo ferroptosis on GPX4 inhibition, we used fluorescence probes to analyse ROS, levels of Fe 2+ and lipid oxidation in senescent cells

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While our dual-pathway strategyintravenous systemic infusion combined with intracerebroventricular (ICV) boostachieved robust hippocampal targeting and synaptic restoration (Fig