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The APRN follow-up at this stage assesses response and determines whether protocol continuation, dose adjustment, or stack modification is appropriate

Costagliola, G

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Despite these diverse origins, such as metabolic dysfunction, immune attack, toxic exposure or vascular injury, the pathophysiological mechanisms involved tend to converge on a common pathway characterized by inflammation, fibrosis, nephron loss and eventual functional failure [15,16,17,18]

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(2015) wherein pkc activation of fak decreases neurite outgrowth, while pkc attenuation has strong neuroplasticity effects via fak-gamma-protocadherin interaction