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ROS accumulation triggers the dissociation of Nrf2 from its cytoplasmic inhibitor Keap1, facilitating Nrf2 nuclear translocation

therefore, it is reasonable to assume that drugs targeting TGR will have a different mechanism of action than praziquantel

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Restraint of proinflammatory cytokine biosynthesis by mitogen-activated protein kinase phosphatase-1 in lipopolysaccharide-stimulated macrophages

then, I-b can bind to NF-B to inhibit its activation and, thus, reduce NSAID-induced intestinal injury (Watanabe et al., 2009)

The result is a market where the least proven compounds are often the loudest, and the best proven ones are undersold