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We further demonstrated that Dlgap5 overexpression promoted NLRP3/CASPASE1 activation and GSDMD cleavage and pyroptosis, while these effects were abolished by the NF-B inhibitor Bay117082 treatment (Figure 5K), establishing that DLGAP5 initiated NLRP3 inflammasome activation via NF-B pathway

[DOI] [PMC free article] [PubMed] [Google Scholar] 122.Kang J.H., Ivey K.L., Boumenna T., Rosner B., Wiggs J.L., Pasquale L.R

Glucose oxidase loaded Cu(2+) based metal-organic framework for glutathione depletion/reactive oxygen species elevation enhanced chemotherapy

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