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They are made of a repeating DNA sequence that protects our vital genetic code from damage

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[30] The extensivity of the -catenin interacting proteins complicates our understanding: -catenin may be directly phosphorylated at Ser552 by Akt, which causes its disassociation from cell-cell contacts and accumulation in cytosol, thereafter 14-3-3 interacts with -catenin (pSer552) and enhances its nuclear translocation

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These include ~50 gene loci, with mutations in three Mendelian genes myocilin (MYOC), optineurin (OPTN) and TANK binding kinase (TBK1) accounting for ~5% of POAG cases with little influence from other genes or environmental factors (12, 20)

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, BPC 159 BPC 157 ,

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[Abstract]2025 Jul 17:43:102164

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Recognition that healthcare systems have historically been designed around male biology, leaving gaps in research, evidence and care

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