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In some DNA tumor viruses (e.g., adenoviruses, papilloma viruses), one specific viral factor (e.g., E4-ORF of adenovirus, ADV, or E6 and E7 of human papilloma virus, HPV-16) may interact with more than one host cell target controlling glucose metabolism

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Once one method is inactivated or exhausted, cancer cells become extremely reliant on the other method to fight against ferroptosis and are supremely sensitive to ferroptosis induced by the other defense system

Although these mice are resistant to diet-induced obesity, they exhibit elevated postprandial triglyceride levels, reflecting dysregulated lipid handling rather than reduced caloric intake

Furthermore, when compared with isogenic wildtype control lines, even a low dose of the inhibitor CB-839 restored the radiation response of the KEAP1/NRF2 mutant cells