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In Huh7, AA suppressed the protective upregulation by OCA or GW4064 of BSEP and SHP, and when STAT3 was knocked down by siRNA ,then the expression was further suppressed

In particular, mitochondrial dysfunction is a key finding in reactive oxygen species (ROS)-induced PD pathology [4,5,6,7]

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These advances not only improve drug bioavailability, but also reduce the frequency of administration, thereby improving patient compliance

MEFs lacking Bmal1 showed lower CI respiration, reduced SAM levels and decreased NDUFS2 methylation (Fig