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doi:10.1111/1471-0528.13574

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Oxalate lowering via AGXT overexpression ameliorates MASH Considering that AGXT is consistently downregulated in MASH leading to hepatic oxalate overproduction in association with disease severity, and that the loss of AGXT accelerates MASH progression in mice 12 , we next sought to determine whether overexpression of AGXT in hepatocytes would improve MASH outcomes by lowering oxalate production

Complete and submit the report Online Regular Mail or Fax: Download form or call 1- 800-332-1088 to request a reporting form, then complete and return to the address on the pre-addressed form, or submit by fax to 1-800-FDA-0178 This recall is being conducted with the knowledge of the U.S

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