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2.6 An integrated framework for UC pathogenesis Overall, the pathogenesis of UC reflects a complex interplay among multiple etiologic factors rather than a single determinant

In a study on breast cancer, ALA-treated MDA-MB-231 and 4T1 breast cancer cell lines both exhibited downregulated expression of EMT markers such as Snail, vimentin, and Zeb1, along with impaired cell migration capabilities [73]

Thus the discovery of cuproptosis reveals that the targets and pathways involved in Cu-dependent cell death are highly correlated with mitochondrial respiration proteins, showing increasing expect for drug discovery

Urgent research is required to identify synergistic combinations (e.g., FSP1 inhibitors with GPX4 inhibitors, ferroptosis inducers with IDO1/m6A inhibitors, and ferroptosis inducers with immune checkpoint blockade) that can simultaneously bypass multiple resistance pathways across diverse GBM subclones

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