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These COX10-deficient oligodendrocytes maintained axonal thickness without a change in the number of oligodendrocyte progenitor cells (OPC) or astrocytes and did not induce microglial activation, suggesting that oligodendrocytes can function without oxidative phosphorylation

p = ns)

For a lab working on neurodegeneration, that focus is exactly what you want to see first

In the latter conformation, the rAmy bypass motif becomes disordered, enabling the receptor ECD to adopt a position similar to CT-bound CTR or AMYR complexes 15

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One proposal is that hypofunction of NMDA receptors (NMDAR) located on inhibitory GABA inter-neurons leads to reduced activity of these inter-neurons, resulting in increased pyramidal glutamatergic neurotransmission and the diverse symptoms occurring in acute psychosis [3]