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Mechanistically, we reveal that VRK2 inhibition disrupts endoplasmic reticulum (ER)-to-Golgi trafficking of SLC7A11, consequently diminishing GSH biosynthesis and predisposing PC cells to ferroptosis

10.1016/j.isci.2024.109135 Summary Keywords tuberculosis, diabetes mellitus, epidemiology, inflammation, multi-omics Citation Araujo-Pereira M, Vinhaes CL, Barreto-Duarte B, Villalva-Serra K, Queiroz ATL and Andrade BB (2024) Intersecting epidemics: deciphering the complexities of tuberculosis-diabetes comorbidity