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Therefore, in the past decade, especially under the promotion of nanotechnology, nanodrugs aimed at depleting GSH or inhibiting GSH biosynthesis have been proposed to improve the effect of tumor treatment and reduce the drug resistance to chemotherapy [10, 13]

We often start higher, then taper to maintenance its a cost-effective way to boost glutathione

when done properly, they are safe but have higher procedural demands

Furthermore, the enrichment of glutathione metabolism and nicotinate/nicotinamide metabolism suggests adaptive responses to oxidative and metabolic stress induced by DHODH inhibition

Several other metabolic targets that modulate ROS toxicity are also being studied preclinically [140, 141]