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Collectively, the existing evidence indicates that multiple oncogenic signaling pathways, such as c-Myc, Kras, and HIF-1, directly regulate key processes in glutamine metabolism, thereby driving the tumor dependence on glutamine

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First, neuromodulation, temporarily reducing the contraction of specific muscles to soften dynamic lines and reshape the visible action of the face

The results of this study confirmed the results from the first absorption site study (Example 10) performed with the peptide SEQ ID NO:3, where maximum absorption was observed in the proximal colon and then in the proximal small intestine of the gastrointestinal tract of the beagle dog

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First, using germ-free animals, directional microbiota transplantation, and gene editing techniques to clarify the causal mechanisms by which protective genera such as Bifidobacterium regulate the hepcidin-FPN1 axis and key molecules of chondrocyte ferroptosis (GPX4, SLC7A11), thereby consolidating the logical chain of microbiota-iron metabolism-cell death. Second, leveraging longitudinal cohorts and multi-omics technologies to reveal the time-dependent evolution of the gut dysbiosis-iron overload-ferroptosis-intestinal dysfunction cycle and identify key windows for early intervention