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These drugs account for 99% of all NSAIDs [26]

Nrf2 binds to and activates antioxidant response elements (ARE), promoting antioxidant gene expression and transcription of FSP1 [140]

[PMID:20494075] Tsai AY, Moroi MK, Les AS, et al

ALA may rescue chemotherapy-induced mitochondrial toxicity to exert neuroprotective effects, but because of the differences in its in vivo experimental findings, future studies of ALA as a potential prevention of CIPN should not be ignored, while other exogenous antioxidant supplements, such as mineral elements, phytochemicals (curcumin and hesperidin), nutritional antioxidants, cytoprotectants and synthetic compounds, have very promising results in basic research

Controlled by IL-6, KRAS has been shown to activate the NF-B pathway during EMT [70], which subsequently regulates downstream effectors, such as transcription factors such as TFIIB, TATA-binding protein (TBP), and CREB binding protein (CBP) [71]

The binding process mainly refers to polar functional groups such as hydroxyl present in either the initial drug or the I-phase metabolite following oxidation