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To restore the redox balance between GSH/GSSG, oral supplementation of the cysteine and GSH precursor, N-acetyl cysteine (NAC), has shown efficacy in protecting photoreceptor survival and retinal function in both animal models and Phase 1 clinical trials when administered orally [12, 13]

In addition, the decrease of KGDHC immunostaining was roughly related to the severity of degeneration