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Excess copper is reduced by FDX1 to generate Cu, which drives cell death via two primary mechanisms: (1) Cu-mediated aggregation of lipoylated TCA cycle proteins (e.g., DLAT) and loss of Fe-S clusters, and (2) Cu-induced disruption of the Npl4/p97 protein quality-control system

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Proc Natl Acad Sci U S A (2012) 109(43):1753742

After cluster generation, the library preparations were sequenced on an Illumina Novaseq platform, and 150 bp paired-end reads were generated

Evaluation of alginate-whey protein microcapsules for intestinal delivery of lipophilic compounds in pigs

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