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Transforming growth factor beta suppresses glutamate-cysteine ligase gene expression and induces oxidative stress in a lung fibrosis model

Interestingly, the main attention on metabolism in the context of HIV has been focused on circulating cells and glucose transporter-1 (GLUT-1), which is upregulated in HIV infected individuals irrespective of ART treatment and is associated with immune activation 65,66

To deplete intracellular GSH, cells were treated with BSO, an inhibitor of glutamyl-cysteine ligase (GCL), the rate-limiting enzyme in de novo GSH synthesis

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Glial cells that provide nutrition to the neurons and provide a link with the immune system, responding to damage by acting as scavengers of pathogens and neuronal debris, were more vulnerable than neurons to organophosphorus pesticides

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