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Additionally, the high expense of these therapies and a variety of other disadvantages prevent the patients from benefiting from them (Jimmy & Jose, 2011)

Many cardiovascular risk factors such as diabetes mellitus, hypertension and hyperlipidemia are demonstrated to stimulate the interaction of Cav-1 and eNOS, reduce nitric oxide production resulting in endothelial dysfunction and subsequently the formation of atherosclerotic lesion 4143

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