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In that manner, NMOSD could also be called an astrocytopathy, along with GFAP-IgG autoimmune encephalitis ( Accordingly, AQP4-IgG was demonstrated to induce IL-6 production and release by astrocytes through the NFkB pathway ( While not considered a direct target of disease as in NMOSD, astrocytes retain central roles in other forms of demyelination

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Quantitatively, mitochondrial abnormalities were significantly higher in aged oocytes but were reduced by uridine supplementation (Fig

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Furthermore, topical application of retinoids on melasma lesions could potentially be associated with secondary hyperpigmentation as a result of drug-induced irritation (Pitowska et al., 2022)