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Redox modifier genes and pathways in amyotrophic lateral sclerosis

Therapeutic targeting of AT ER stress shows promise through chemical chaperone interventions

It has been argued that in human cells, NMD is restricted to a pioneer round of translation initiated on mRNAs associated with the cap-binding complex (CBC) and that the exchange of the CBC for the eIF4F translation initiation complex renders mRNAs immune to NMD

Pancreatic cancer: Advances and challenges

10.1016/j.freeradbiomed.2018.11.002 134

Targeted gene deletions in mouse models highlight their unique functions - leptin knockouts develop severe obesity with decreased energy expenditure, while mice lacking adiponectin exhibit profound insulin resistance