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Acknowledgments We thank Ping He for plasmid constructs, and Andrew Palmer, Rafael Loureiro and members of the Shippen lab for helpful discussions

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Additionally, it is suggested that a subset of children with ASD have automatic nervous system dysfunction leading to greater GI instability [37]

A cellular NF-B regulates the expression of numerous immune system components and modulates inflammation (Li and Verma 2002)

These results also indicate a change in the expression of synaptic dopamine receptor proteins (D1 and D2), a decreased in serotonergic receptors (5-HT1A and 5-HT2A), metabotropic GABA-B, and muscarinic receptors (18)

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