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As explained above, engagement of AGE with RAGE generates inflammatory response via activation of cdc42 that ultimately upregulates Rac1

This aligns with evidence that mitochondrial fission can proceed without changes in DRP1 expression, such as with deletion of PTEN-induced kinase 1 or protein kinase A activation, which affect DRP1 phosphorylation at Ser616 without altering protein levels [61, 62]

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Antioxidants (Basel) (2020) 9:1070

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