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[DOI] [PubMed] [Google Scholar] 89.Samanta D

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Additionally, ferrochelatase 1 has been identified as a key effector molecule in cuproptosis, driving the cell death proce ss by regulating protein acylation modifications as well as degrading iron-sulfur cluster proteins ( SLC31A1 expression, resulting in mitochondrial copper overload, which subsequently activates the cuproptosis pathway (Yu et al., 2025)

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