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Furthermore, norepinephrine has the potential to promote the synthesis of pro-inflammatory cytokines by upregulating the activity of Nuclear Factor Kappa B ( NF-B ) through key inflammation- or proliferation-related pathways such as those of JNKs and p38-MAPKs

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References 1 AdessoS.MagnusT.CuzzocreaS.CampoloM.RissiekB.PacielloO.et al

This cell death form depends on iron rather than other metals, and can be suppressed by iron chelator deferoxamine (DFO)

Our analysis revealed a positive correlation between the NF-B pathway and EAAT3 expression in the KP model (Supplementary Fig

To mechanistically determine whether 15LO1 pathway activation alters the intracellular redox balance, particularly under T2 conditions, these ex vivo findings were modeled in vitro in primary HAECs