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Chelating this zinc signal with the membrane-permeable chelator TPEN effectively blocks the activation of LPS-induced signaling pathways, including the p38 MAPK, extracellular-signal regulated kinases 1/2 (ERK1/2) and NF-B pathways, ultimately inhibiting the release of proinflammatory cytokines such as TNF- [18]

High concentrations of acetate and butyrate have been shown to alter tight-junction proteins and histone acetylation patterns in cervical epithelial cells, potentially facilitating viral entry and supporting HPV persistence