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This model suggests that, beyond TG accumulation and inflammation, additional interrelated mechanisms such as lipotoxicity, endoplasmic reticulum (ER) stress, mitochondrial dysfunction, and inflammasome activation collectively contribute to the development and progression of MASLD

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The red blood cell issues and Shungus findings suggesting that narrowed blood vessels are creating a hypoxic or low oxygen environment in the brain provide another possible way to explain for the low energy problems in ME/CFS