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However, excessively high ROS levels can cause severe damage to nucleotides, proteins, cell membranes, and organelles, ultimately leading to cell death

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Nitric oxide and nitrotyrosine in the lungs of patients with acute respiratory distress syndrome

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M+2 acetyl-CoA uptake assay in immunopurified ER ER were purified as described above from SLC33A1 knockout HEK293T expressing EMC3-mScarlet-3xHA (ER-tag) complemented with a vector control or SLC33A1 cDNA

Following HMGB1 inhibition or 5-HT7R knockout, the downstream Nrf2/xCT/GPX4 signaling pathways was upregulated, resulting in alleviated depressive-like behaviors (Du et al., 2025)