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This review also summarizes and highlights current limitations in targeting the Th17/Treg balance for AR treatment and suggests future research directions, aiming to provide new insights and strategies for clinical management (Fig

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We employed LPS-induced septic shock models, both with and without reconstitution with SLC25A33-silenced BMDMs, alongside a CLP-induced sepsis model, to examine the role of SLC25A33 in the inflammatory responses in vivo