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This may necessitate a corresponding decrease in the consumption of fat prior to training and competition, to avoid excess energy intake and ensure athletes are able to meet carbohydrate requirements for exercise

The disease is caused by a mutation in the ATP7B gene, which codes for a protein that facilitates the incorporation of copper into proteins (such as ceruloplasmin) and also the transportation of copper into vesicles that allow it to be secreted in bile.1 The critical effect of a mutation in ATP7B is diminished copper secretion into bile, which leads to excess copper accumulation in the hepatocyte

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Theres no recommended upper limit for B12, so overdosing is not a concern

The application of L-asparaginase as a plasma Gln-depleting agent with a clear therapeutic effect in solid tumors warrants further investigation