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As such, it is possible that redox-associated extrahepatic mechanisms may contribute to the activation of the hepatic AMPK pathway

They were first identified in 1994 in leukemic cells with the CD34+CD38 phenotype [22] and have since been identified in different types of cancer

The GR protein has a modular structure similar to other members of its nuclear receptor family

The KD resulted in the reduction in muscle mass, integrated by decreased plasma insulin and IGF-1 levels, and elevated plasma corticosterone levels

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