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These findings underscore the key role of high GGT expression in promoting drug-resistant phenotypes in tumor cells

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The gut microbiota metabolite glycochenodeoxycholate activates TFR-ACSL4-mediated ferroptosis to promote the development of environmental toxin-linked MAFLD

Some studies suggest that the resistance mechanisms of c-MET inhibitors usually fall into one of two categories: (i) target resistance mediated by secondary MET kinase domain mutations and/or allele amplification of METex14

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