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In addition, microbial overgrowth impaired the uptake and metabolism of PUFAs and other molecules

Previous studies have shown that a SEPHS2 mutant in which Sec is replaced by cysteine (SEPHS2 U/C) rescued the SEPHS2-deficient phenotype 42,43 (Extended Data Fig

These processes share plasma membrane disruption, resulting in intracellular component release into the interstitial space, which amplifies local inflammation, disrupts immune homeostasis, and worsens tissue damage [41]

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