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Since activation of cMyc expression via CRISPRa as well as cMyc T58A expression was sufficient to sensitize SCLC cells to ferroptosis in our experiments, it is tempting to speculate whether NOTCH pathway activation by cMyc might be responsible for this

Gronniger, E

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Furthermore, the saturation threshold effect was evaluated separately in subgroups stratified by BMI and age

AKT can enhance FOXM1 expression and form a positive feedback to enhance venetoclax resistance in FLT3 wild-type AML (67)